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Anisimov 2013

doi: 10.3389/fneur.2022.1063803 21 BerdyskiM.MisztaP.SafranowK.AndersenP

Significantly, by downregulating the pro-apoptotic protein Bax and upregulating the anti-apoptotic protein Bcl-2, the combination treatment altered apoptotic signaling and may have a protective impact against hepatocyte death, a crucial mechanism in the development of NAFLD