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The primary mechanical injury in TBI triggers secondary injury cascades involving neuroinflammation, oxidative stress, mitochondrial dysfunction, and excitotoxicity

L., Latz, E., Ontiveros, F

Peer-reviewed molecular research has shown that maintaining high levels of cellular NAD+ influences inflammatory pathways through several distinct biological mechanisms: Higher intracellular NAD+ levels directly lower the overall cellular production of IL-1, IL-6, and TNF-