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Defective mitochondrial function and impaired energy metabolism are highly implicated in the pathogenesis of PD [8]

We have selected the chronic rotenone-induced cellular model of PD because in this model, damage develops over a protracted time course and features of PD pathogenesis, such as modest systemic complex I dysfunction (a 25% loss of activity), oxidative damage, and -synuclein accumulation are reproduced [17]

Step 2: Fat Utilization L-Carnitine ensures these fatty acids are transported into mitochondria and converted into energy