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Rat experiments demonstrated enhanced regeneration of uterine and testicular tissue following induced damage, with histology highlighting reduced fibrosis and enhanced vascular repair

ROS cause lipid peroxidation, DNA damage, and protein denaturation, which directly impair cell functions and activate pro-inflammatory signaling pathways (e.g., NF-B and Nrf2) (Muro et al., 2024), exacerbating inflammation and creating a vicious cycle between inflammation and oxidative stress

TEV protease cleaves between Q (Gln) and G (Gly) or S (Ser)

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