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This suggests that prophylactic or early intervention with anti-inflammatory agents that block upstream pathways like NF-B or p38 MAPK following a known insult (noise trauma) could potentially decouple the inflammatory response from the triggering event, preventing maladaptive neural plasticity and the development of tinnitus

This damage can be mitigated by the cellular antioxidant defense mechanism known as mitochondrial autophagy [139]

Given that the SLC39A8 p.Ala391Thr (rs13107325) variant has been associated with more than 22 clinical conditions, an important question is how this single missense variant can cause such a wide range of pathologies 44

This transition from discrete to continuous sampling has reshaped design constraints: sensors must maintain electrochemical stability within complex biological matrices and ensure biocompatibility over extended wear [27,28]