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Individuals with GS exhibit increased bilirubin production from both hepatic and erythroid sources, with evidence of elevated hepatic haem production and occasional haemolysis[13]

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Of particular interest was another clinical observation that a WD patient with the G54S PRNP variant presented with very severe symptoms, while his older sister, who carried the same ATP7B genotype but was clinically asymptomatic, lacked this variant allele 22

Phys Ther Sport 14(2):124130 Zayni R, Thaunat M, Fayard JM et al (2015) Platelet-rich plasma as a treatment for chronic patellar tendinopathy: comparison of a single versus two consecutive injections