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This modified peptide that lacked Tyr at residue 10 would be predicted not to lead to cell death or protein oxidation of neuronal cultures or be able to activate the nitrone spin trap to form the EPR-measurable nitroxide free radical if the Tyr residue of A42 was critical to these effects of A42

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Conclusion: In this prospectively followed AIH cohort, the severity of liver disease and stage of fibrosis did not differ in chronic AIH patients based on IgG levels

A0A3Q0F2H5 (ankyrin repeat-containing protein BDA1-like, 451 amino acids) functions as a membrane-associated signalling scaffold that regulates stress-induced defence and programmed cell death