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In stimuli-responsive drug delivery systems, a trigger is employed to selectively separate drug molecules from their carrier, thus mimicking the in vivo pulsatile release of several types of physiological chemicals, such as hormones, like estrogen and insulin (Murdan, 2003

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Activating Nrf-2 signaling depresses unilateral ureteral obstruction-evoked mitochondrial stress-related autophagy, apoptosis and pyroptosis in kidney

The amount of O 2 produced by pyruvate and malate/glutamate metabolism can be amplified by rotenone, a Complex I inhibitor (Muller et al., 2008)