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Eight proteins play critical roles in RCC with bone metastasis via mitochondrial dysfunction

Egner PA, Stansbury KH, Snyder EP, Rogers ME, Hintz PA, Kensler TW

[4] The proposed mechanism of action, based only on preclinical animal studies, involves potential modulation of growth factor pathways, particularly vascular endothelial growth factor (VEGF), which may promote angiogenesisthe formation of new blood vessels