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If verified in humans, this would position epitalon not merely as a telomere compound, but as something operating at the level of epigenetic clocks the same biological machinery measured by tools like the Horvath clock to determine biological age

Accordingly (and against accepted convention), the contribution of CYP2E1 to ALD pathophysiology may be more complex than originally thought and is likely dependent on the cellular microenvironment or subject to modulation by other signaling pathways 15,37

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