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It was confirmed that IL-33 served as an alarmin released by stressed hepatocytes, which could bind to the receptor termed suppressor of tumorigenicity 2 (ST2), then activated NF-kB and mitogenactivated protein kinases (MAPKs) pathways, ultimately promoting the formation of liver fibrosis (100, 101)

This claim requires significant caution, as long-term human data is not available to confirm durability of results

Studies have shown that hepcidin can enter the central nervous system through the BBB (Raha-Chowdhury et al., 2015), which, with increased permeability after TBI, may allow a large amount of hepcidin to the brain tissue

According to these findings, spirulina promoted wound healing by increasing the number of myofibroblasts