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49, 159172.10.1111/j.1365-313X.2006.02938.x 51 PasternakM.LimB.WirtzM.HellR.CobbettC

Oncogenic mutations, such as those in KRAS, EGFR, TP53, LKB1, and KEAP1, drive these alterations through distinct mechanisms affecting glucose metabolism, mitochondrial function, redox homeostasis, and nutrient sensing

Consistent with this result, the pharmacological inhibition of mTOR activity in the brain of AD mice was shown to restore autophagy and to reduce A and Tau levels (370373)