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Acsl4 dictates ferroptosis sensitivity by shaping cellular lipid composition

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6.2.5 Implication for MFH These genetic axes motivate hypotheses that MFH actives could: (i) support mitophagy/mitochondrial quality control, (ii) reinforce endogenous redox buffering (e.g., Nrf2-GPX4), and (iii) dampen inflammation linked to mitochondrial stress