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SCFA abundance was altered in several diseases (81)

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There are numerous potential therapeutic targets, such as the development of targeted inhibitors of ion channels (sodium channels, potassium channels, calcium channels, etc.) and glutamate receptors, to reduce the neuronal excitotoxicity caused by excessive glutamate release (52, 53)

Similar to the positive-regulated genes, our qPCR validation on negative-regulated genes (Figure 2C) were also matched well with the transcriptomic data (Figure 2A