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Social interaction deficits parallel evidence that prenatal cannabinoid exposure disrupts sociability via altered endocannabinoid and oxytocin signaling [54, 55, 56], while broader affective and motivational abnormalities align with persistent synaptic dysregulation in corticolimbic circuits following early-life adversity [57, 58, 59]

Also, a phase III randomized placebo-controlled trial demonstrated that another compound protective against oxidative stress, dexpramipexole, did not improve motor function in ALS patients (Cudkowicz et al., 2013)

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