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Description

Here, we demonstrate that genetic disruption of iSPNs function by Ntrk2/Trkb deletion in mice results in increased striatal dopamine and midbrain dopaminergic neurons, preceding hyperkinetic dysfunction

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Furthermore, this association is also observed in scavenger receptor A expression, leading to the formation of foam cells [32,33,34]

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The participation of ascorbate in the aforementioned processes is partially based on its anti-antioxidant and anti-inflammatory properties

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