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Although synthesis of reactive oxygen species and production of reactive oxygen species were present in both WT and Cth / BMmacs, importantly, the metabolism of reactive oxygen species was not present in the pathways induced by H

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H 2 S production, measured using the 7-AzMC fluorogenic probe as previously described (Figures 2E, D YRK1A expression in HepG2 cells induced a significant increase in H 2 S production without affecting cell proliferation and/or viability (Supplementary Figure 1C), whereas two mutant forms of DYRK1A (p.S324R and p.S311F) that had lost their kinase activity ( 2 S production (Figure 2E)