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Clinical implications and mechanistic insights of ischemia-reperfusion injury, particularly in the liver, have been extensively reviewed [4]

However, the analysis did not show a significant separation between groups with the set of variables used as predictors (OCPs concentrations and oxidative stress indicators) (Wilks' Lambda = 0.44

InsR-B, which contains 12 amino acids encoded by exon 11 of InsR, mediates the metabolic actions of insulin such as the uptake of glucose by muscle and adipose [25, 28]