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Therefore, understanding the cellular and molecular mechanisms that contribute to chronic radiodermatitis onset and progression is essential for developing targeted therapies that can prevent or reduce the severity of fibrotic changes, ultimately aiming to preserve patient quality of life and improve cancer care post-radiotherapy

Efflux transporters that transport drugs out of the enterocytes can limit the oral bioavailability of their drug-substrates

371 In osteoblasts, the administration of GLP-1 and GIP incretins suppressed the excessive expression of the pro-degradative enzymes MMP-3 and MMP-13 induced by IL-1 stimulation

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