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-Sitosterol selectively inhibits the activity of the pro-inflammatory mediator COX-2 and reduces the levels of the oncogenic PGE 2 in tumor tissues, blocking the promotion of tumor cell proliferation by the COX-2/PGE 2 inflammatory axis (92)

Although inflammation is reflected in selective biomarkers, traditional inflammatory markers such as C-reactive protein (CRP) and erythrocyte sedimentation rate have also shown an increased trend in ME/CFS, particularly in those with mild/moderate disease generally not raised in ME/CFS (226)