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In conclusion, these data show that loss of ATP6AP1 impairs lysosomal function and that changes in lysosomal pH and size of acidic compartments directly affect cytosolic pH, contributing to the resistance to mitochondrial dysfunction associated with m alterations

Transglycosylation of disaccharides, glycosyl fluorides, aryl galactosides f

found that the N protein can undergo phase separation with TAK1 and IKK, concurrently suppress stress granule formation, promote NF-B activation, and ultimately trigger cellular pyroptosis (Fig