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demonstrated that MSCs transferred mitochondria to tumor cells via microvesicles in response to oxaliplatin treatment, restoring mitochondrial function and suppressing mitophagy, thereby enhancing chemotherapy resistance [344]

Two highly expressed genes encoding glutathione S-transferase (Gstk1 and Gstm) were downregulated in hypertrophic hearts, importantly because glutathione S-transferase plays the role of an antioxidant by conjugating glutathione on various substrates to protect the heart from oxidative stress (Yan et al., 2012)

Icariin alleviates cisplatin-induced premature ovarian failure by inhibiting ferroptosis through activation of the Nrf2/ARE pathway

There is strong evidence linking elevated ROS levels to cancer progression (Yang and Chang, 2018) and transmembrane receptor tyrosine kinase family (Singal et al., 2019), can become abnormal through gene amplification, mutation, or overexpression