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The decreased expression of pro-inflammatory factors, including IL-1, TNF-, CD86, and iNOS, and the increased expression of anti-inflammatory factors, including IL-10, TGF-, CD206, and Arg-1, clearly reflect the anti-inflammation of CuZnHis assemblies, which have the potential to treat oxidative stress-related diseases

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By integrating these strategies, we can move toward a new generation of interventions that not only slow disease progression but may also mitigate the impact of Alzheimers disease across diverse genetic backgrounds, heralding a future with more effective, targeted treatments

Nature of tau-associated neurodegeneration and the molecular mechanisms