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The main source of oxidative stress in the genesis of PD is the activation of glial cells and dopaminergic neuronal death of the substantia nigra [36, 37]

As a result, there remains a substantial gap between laboratory findings and clinical application

Supplementation of vitamin E exceeding 400 mg/day in patients with cystic fibrosis has demonstrated deleterious effects, including an elevated risk of mortality in adults (184) and occurrence of hemorrhagic stroke (185)