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Co-treatment with the ferroptosis inhibitor ferrostatin-1 (Fer-1) or the mitochondrial-targeted antioxidant SS-31 markedly suppressed high glucoseinduced accumulation of 4-HNE and MDA, thereby mitigating lipid peroxidationdriven cellular injury (Xiong et al., 2024)

Consistent with these findings, HIF-2 knockdown in renal cell carcinoma inhibited the expression of CXCR4, a chemokine receptor used as a renal cell carcinoma stem cell marker, resulting in the inhibition of spheroid and tumor growth 150