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Taken together, these results suggest that HDAC3-mediated DUSP5 downregulation may promote the progression of diabetic cardiomyopathy

The ability to form sedentary communities or biofilms in anatomical sites, such as the middle ear, has also been shown to contribute to the in vivo persistence of NTHi (Jung and Do, 2013

Genetic and pharmacological induction of Nix was able to reestablish mitophagy in PINK1 - and PRKN -related PD patient cell lines, and Nix over-expression resulted in an improvement in mitochondrial ATP production