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Description

This metabolic reprogramming arises as a consequence of mitochondrial damage and a hypoxic microenvironment and initially serves as an adaptive response to energy stress

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Additionally, this innovation would also benefit the pharmaceutical industry

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Data availability The inclusion and exclusion criteria data that support the findings of this study are available from ClinicalTrials.gov The synthesized raw data is also available upon request

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NFKB induces E- selectin (also known as ELAM-1 ) on the surface of vascular endothelial cells

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