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Retrieved 4 January 2015

In animal studies, Semax significantly boosts brain-derived neurotrophic factor (BDNF), a protein that supports neuron growth and learning

Therefore, glucose transporters and glycolytic enzymes could be a potential therapeutic target to modulate chondrocyte metabolism to treat OA ( GLUT1 and other glycolytic enzyme expression, potentially promoting anaerobic glycolysis in OA cartilage chondrocytes and enhancing cell vitality