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The pharmacogenetics of metformin and its impact on plasma metformin steady-state levels and glycosylated hemoglobin A1c

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Excessive ROS are produced in physiological functions such as mitochondrial function and phagocytic activity

This metabolic inflexibility in Insr P1195L/ + mice could be also due to compensatory increase in fat utilization in fat and skeletal muscle during dark phase caused by impaired glucose uptake in these tissues