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IB degradation exposes the nuclear localization signal on NF-B, thus allowing the nuclear translocation of the subunit and activation of the transcription of its target genes (Fig

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As the mice in that study were kept in a standard animal holding facility (not specific pathogen free), perhaps exposure to infectious agents may have contributed to this early, peripheral deposition of A (which likely would be much more pronounced in an Alzheimers disease model than in wild-type mice)

p53 fl/fl lung cancer model, where J10 treatment significantly extended overall survival and markedly delayed disease progression (Fig