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Both preclinical and clinical studies are now revealing that to optimize anti-tumor effects one may require (a) an agent to activate T cells directed against a tumor antigen or antigens, (b) potentiating the immune response with the use of agents such as cytokines, (c) reducing or eliminating immunosuppressive entities systemically or in the TME, and (d) altering the phenotype of the premalignant or tumor cells to make them more susceptible to immune-mediated lysis [141]

33,34 After oral administration, absorption of glutathione is very poor mainly due to hydrolysis by intestinal and hepatic -glutamyltransferase as mentioned above, and for that reason, most therapeutic approaches using the oral route struggle to achieve results and the patients seek less safe treatments like intravenous treatment often under uncontrolled situations which may not be medically supervised, in some countries

doi: 10.1016/j.lfs.2019.03.053

However, microglial activation can become detrimental under pathological conditions