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Novel cancer therapy: targeting methionine metabolism

It works by binding to the ghrelin receptor (GHS-R1a) in the hypothalamus and pituitary, triggering a calcium/PKC signaling cascade that releases stored growth hormone, while separately blunting somatostatin's inhibitory signal

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Since up-regulation of SCN3A has been demonstrated to enhance neuronal excitability and contribute to the development of epilepsy, the reduction of SCN3A expression by VPA due to its promotion on methylation may underlie the anti-epileptic effect of VPA