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Rather, GLP-1 receptor activation results in cell growth, and the enzyme release may simply reflect adaptive growth instead of an inflammatory response

finds that inhibition of SLC7A11 after Erastin treatment increases radiosensitivity in SLC7A11 + breast cancer cells in vitro and in vivo , while it has no effect on SLC7A11 - cancer cells, this process accompanied by a decrease in intracellular GSH synthesis that promotes cell death (Ye et al., 2020)

In addition, researchers observed in vitro that FDX1-mediated Cu metalorganic frameworks (CuMOF) release Cu ions and DLAT in KRAS -mutant NSCLC, thereby inducing proteotoxic cell death and achieving effective anti-tumor effects (Li K

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