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Impaired synaptic plasticity (i.e., long-term potentiation, long-term depression, paired-pulse facilitation) and deficits of synaptic transmission underlie these phenomena and have been reproducibly demonstrated in aged rodents compared to their younger counterparts (Norris et al., The Hippocampal Proteome with Learning and Memory Given that age-related decrements in cognitive function occur heterogeneously in both human and animal model populations, and that cognitive decline occurs on a background of aging-related proteomic alterations, studies pairing behavioral assessments of cognitive function with quantitative proteomic analyses at multiple ages are needed

Sezen, S

The FDA usually relies on clinical trials when it is looking at adding a substance to the 503A bulks list, which determines what ingredients compounding pharmacies can use in mixing drugs

USA 108 , 1961119616 (2011)