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Recently, Chen and colleagues developed a SAD cortical organoid model from human iPSCs and exposed it to serum to mimic BBB breakage, a common AD feature, and observed increased A-like pathology, hyperphosphorylated tau, synaptic loss, and an impaired neural network [191]

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Interestingly, the results revealed that all four components inhibited both the monophenolase and diphenolase activity of tyrosinase, which had smaller molecular weights, correlating with greater tyrosinase inhibitory activity

Wiley Blackwell, pp 255281 Salvia-Trujillo L, Qian C, Martn-Belloso O, McClements DJ (2013) Modulating -carotene bioaccessibility by controlling oil composition and concentration in edible nanoemulsions