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These results suggest that HU attenuates kidney injury in STZ-induced diabetic mice by inhibiting the abnormally activated mTOR signaling pathway

Although alterations in genetic factors have been linked with its etiology, exponential accumulation of environmental entities such as reactive oxygen species (ROS) initiate a cyclic chain reaction resulting in accumulation of cellular inclusions, dysfunctional mitochondria, and overwhelming of antioxidant machinery, thus accelerating disease pathogenesis