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As mentioned previously, several studies have reported that brain GSH levels are reduced following TBI and genetic variations in GSH-dependent, peroxide/electrophile-detoxifying enzymes, such as glutathione-S-tranferase-4 and glutathione peroxidase-1, can sensitize mice and rats to brain injury induced by TBI (Tyurin et al., 2000

Although effector T cell responses are generally potent, a subset of infectious diseases and tumours have evolved a large variety of escape mechanisms to bypass T cell control 1

A study comparing ID and subcutaneous (SC) delivery of radiolabeled vitamin B12 in rabbits found that while the bioavailability was slightly higher for the SC route, the Tmax (time to reach maximum concentration) was almost identical for both methods