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This further supports the notion that promoting complete mitophagic flux, rather than just the initiation of mitophagy, could be critical in mitigating pancreatic inflammation and damage in AP

This overlap suggests that FOXO4s activation by oxidative stress and inflammation might follow similar mechanisms, pointing to a broader role of FOXO4 in stress and immune response contexts

If we consider that Cd stimulates autophagy, the increase in the level of SQSTM1 remains surprising but could be explained by the fact that Cd activates ERK1/2 in cells with very low level of p-ERK1/2, and that RAF1/Raf-MAP2K/MEK-MAPK/ERK signaling can stimulate SQSTM1 transcription (Klionsky et al

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