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Additionally, increasing studies have revealed the important mechanisms in the communication of mitochondria with other parts of cells (16), including cytochrome c release to induce cell death, 5-adenosine monophosphate-activated protein kinase activation to control mitochondrial fission and fusion, ROS generation to activate transcription factors, and mtDNA release to activate immune responses (Figure 1)

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The increase in AA-induced by various factors and the activation of inflammatory cells promote ferroptosis by impairing mitochondrial function and generating large amounts of free radicals