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Originally identified during investigations involving sleep physiology and neurological regulation, DSIP peptide has attracted substantial scientific interest due to its unique structure and potential interactions with biological systems associated with sleep cycles and neuroendocrine signaling

In the granulosa cells of patients with PCOS and in letrozole-induced rat models, the HIF-1/BNIP3 mediated mitophagy is suppressed, resulting in a decrease in the clearance capacity of damaged mitochondria and accumulation of ROS, thereby inducing mitochondrial dysfunction [71]

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