slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells links GSH sensing with iron metabolism: Molecular Cell DLAT sustains redox homeostasis and

Name: slc25a39 is necessary for mitochondrial glutathione import in mammalian cells links GSH sensing with iron metabolism: Molecular Cell DLAT sustains redox homeostasis and
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DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39 mediated mitochondrial glutathione transport ScienceDirect Glutathione driven redox decisions in cell survival and death Development of a mouse model expressing a bifunctional glutathione synthesizing enzyme to study glutathione limitation in vivo Journal of Biological Chemistry Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis bioRxiv

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doi: 10.1161/01.res.0000035038.41739.cb

Estradiol protects against ATP depletion, mitochondrial membrane potential decline and the generation of reactive oxygen species induced by 3-nitroproprionic acid in SK-N-SH human neuroblastoma cells