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Next, we used surgical forceps to grip the BPCDX, which was then inserted into the intrastromal pocket through the access cut 65

In the absence of ovarian dysfunction and insulin resistance in mice, high dietary fat intake increases the risk of endometriosis (69)

This suggests that the cellular stress signature probably drives both the death of resident KCs and the generation of monocyte-derived KCs

Another observational study by Ishibashi et al., demonstrated that comparatively to epalrestat, 500 nM ranirestat inhibited the effects of high glucose on elevated sorbitol levels, vascular cell adhesion molecule-1 mRNA levels in umbilical vein endothelial cells, and THP-1 cell adherence to human umbilical vein endothelial cells [138]