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10.1101/gr.144700 12 BarbosaG

A notable paradox is that genetic ablation of the MCU, which markedly reduces mitochondrial Ca 2+ uptake and stimulusmetabolism coupling in PACs, does not significantly decrease the severity of experimental AP ( 2+ overload remains mechanistically important, but acts within a broader and partially redundant injury network that also includes extramitochondrial Ca 2+ sources, residual bioenergetic support from glycolysis, ROS generation, and inflammatory amplification

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