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The immune response to this damage, characterized by M1 macrophage polarization, neutrophilic inflammation, and Th17 activation, further exacerbates lipid peroxidation and ferroptosis, creating a vicious cycle that drives disease progression

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Charoenwongpaiboon T, Punnatin P, Klaewkla M, Pramoj Na Ayutthaya P, Wangpaiboon K, Chunsrivirot S, Field RA, Pichyangkura R (2020) Conserved calcium-binding residues at the Ca-I site involved in fructooligosaccharide synthesis by Lactobacillus reuteri 121 inulosucrase