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Oxidative stress is a pivotal factor in the pathophysiology of DMD that contributes significantly to muscle degeneration [4,5,6]

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Based on T4 research, TB-500 has been suggested to exert potential action on cellular motility via elevating the levels of actin proteins

Moreover, the gene expression profile of antioxidant enzymes showed a significant increase in SOD and CAT via the dermal route and GR and GPx via the subcutaneous route [99]