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Defects in glutamate signaling are implicated in autism, but how such defects affect neuronal signal processing and cause varied autistic phenotypes remains unknown [6]

fragilis can release a unique pro-inflammatory LPS-subtype and fragilysin, leading to an impaired intestinal paracellular and transcellular epithelial barrier, thereby allowing an already leaky barrier a gateway for the enhanced entry of microbiome-derived neuro- and entero-toxins into the circulation (Lukiw et al., 2021)

Among the ALOX isoform, overexpression of ALOX5, ALOX12, and ALOX15 significantly sensitize cells to ferroptosis [50]

Trionfini and Benigni 2017)