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Chip 23 , 51515164 (2023)

found that the mechanism of selenium inhibition of tumor cell cycle progression may be related to selenium regulation of RNA expression of mitotic cell cycle protein B and cdc2 kinase activity by analyzing the cell cycle effects of selenoethanethionine in colon cancer cells in their laboratory (157)

Histopathological changes in the acute, subacute, and chronic stages emphasize the evolutionary nature of AA and the importance of early intervention

Crosstalk between peroxisome proliferator-activated receptor gamma and the canonical wnt/-catenin pathway in chronic inflammation and oxidative stress during carcinogenesis