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NADPH contributes to recycling in oxidized antioxidant systems involving glutathione, thioredoxins, glutaredoxins, and peroxiredoxins and maintains another reactive oxygen species (ROS)-detoxifying enzyme, catalase, in its active form, suggesting that the homeostatic level of G6PD-derived NADPH determines the rate of oxidative damage [8]

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In the gut, their classical role was attributed to protection against parasitic infectionsa notion supported by the Th2-dominated responses (IL-4, IL-5, IL-13) and elevated eosinophil counts observed in helminthiasis ( One shift in perspective is the recognition that intestinal eosinophils are normal residents with homeostatic roles

These effects lead to more enhanced chemotherapy efficiency in a murine model of lung cancer 55 , underscoring the potential of NRF2 activation not only in widening the therapeutic window but also in actively reshaping the immune landscape within the TME