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Medications can reduce the pain of peripheral neuropathy. Tesamorelin has never been directly studied for this purpose, but research into therapies based on growth hormone indicates it might be useful: Growth hormone (GH) and insulin-like growth factor-1 (IGF-1) have been shown to accelerate axonal regeneration, maintain proliferating Schwann cells, reduce muscle atrophy and thereby improve motor and sensory functional recovery following peripheral nerve injury in a number of studies

Tesamorelins visceral-fat effect rests on randomized, controlled human trials the top of the evidence hierarchy which is why it earned an FDA approval and a real prescribing label

Studies suggest this selectivity may be related to ipamorelins binding kinetics and partial agonist activity at GHSR it may preferentially activate GH-releasing conformational states of the receptor

The case for co-administration is not that they fail apart