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Although no clinical trials have currently utilized these medicines for RA, the common molecular irregularities-such as p53 mutations, oxidative DNA damage, and neovascularization-indicate that p53-modulating approaches merit exploration in RA models and, eventually, human investigations

Activation of nicotinamide adenine dinucleotide phosphate (reduced form) oxidase by advanced glycation end products links oxidative stress to altered retinal vascular endothelial growth factor expression

For example, footprints for AP-1 subunits and other TFs associated with time on ART were identified in the core promoter of important genes mediating immune and senescence phenotypes

Zhou et al., 2020) and hepatocellular carcinoma (Lv et al., 2018) causing growth arrest and apoptosis, however, in these studies the effect on 5hmC/5mC levels at these gene loci was not measured